Ki-Hyuk Shin, MS, PhD
Dr. Ki-Hyuk Shin's major research activities focus on the role of DNA damage and repair in oral carcinogenesis, the role of telomerase activity in oral carcinogenesis and replicative senescence of normal human oral cells.
Shin KH, Kim RH, Kim RH, Kang MK, Park NH. hnRNP G elicits tumor-suppressive activity in part by upregulating the expression of Txnip. Biochem Biophys Res Commun. 2008 Aug 8;372(4):880-5. Epub 2008 Jun 9.
Shin KH, Kim RH, Kang MK, Kim R, Kim S, Lim PK, Yochim JM, Baluda MA, Park NH. p53 promotes the fidelity of DNA end-joining activity by, in part, enhancing the expression of heterogeneous nuclear ribonucleoprotein G. DNA Repair. 2007; 6:830-840.
Shin KH, Kang MK, Kim RH, Christensen R, Park NH. Heterogeneous nuclear ribonucleoprotein G (hnRNP G) demonstrates tumor suppressive effect against oral squamous cell carcinoma cells. Clinical Cancer Research. 2006; 12:3222-3228.
Shin KH, Kang MK, Kim RH, Kameta A, Baluda MA, Park NH. Abnormal DNA end-joining activity in human head and neck cancer. International Journal of Molecular Medicine. 2006.17:917-924.
Shin KH, Kang MK, Dicterow E, Kameta A, Baluda MA, Park NH. Introduction of human telomerase reverse transcriptase to normal human fibroblasts enhances DNA repair capacity. Clinical Cancer Research. 2004; 10:2551-2560.